Kev ua txhaum ntawm cov txheej txheem ntawm tsim ntawm bile thiab nws cov khoom ua rau ua rau ib qho tsis txaus noj ntawm biological tshuaj mus rau hauv lub bile ducts. Qhov kev mob no, intrahepatic cholestasis, nrog rau kev kho mob tsis tu ncua tsis ua rau muaj kev phom sij. Txawm li cas los xij, qhov ntev ntawm cov pathology yuav provoke lwm yam kab mob loj.
Ua rau ntawm intrahepatic cholestasis syndrome
Factors contributing rau lub deterioration ntawm bile synthesis:
- hypothyroidism ;
- sepsis;
- noj tshuaj;
- Alagill's syndrome;
- kab mob siab;
- chromosomal ntshawv siab;
- kev xeeb menyuam ntawm cov metabolism hauv;
- endocrine kab mob;
- hormonal tsis txaus nyob rau hauv cev xeeb tub;
- kev quav dej caw;
- cirrhosis ntawm lub siab;
- kev qaug;
- kab mob bacterial infections;
- plab hnyuv dysbiosis;
- endotoxemia.
Cov tsos mob thiab cov tsos mob ntawm tus mob cholestasis
Rau thaum ntxov mob tshwm sim ntawm cholestatic syndrome muaj xws li pruritus thiab jaundice.
Lub ntsiab symptomatology:
- fecal decoloration;
- darkening ntawm zis;
- kev loj hlob ntawm daim siab loj;
- "Rog" quav;
- dyspeptic ntshawv siab;
- zoo nkaus li kev chim siab hauv hom lus;
- qee zaus - mob lossis hnyav hnyav hauv txoj cai qis dua.
Kev kho mob ntawm intrahepatic cholestasis
Txoj kev kho ntawm cov tau piav qhia txog tus kab mob no yog xav kom tshem tawm qhov kev ua cholestas.
Tib lub sij hawm, kev kho mob yog muab los pab rau kom tsis txhob muaj cov tsos mob ntawm txhua kis. Kev teem caij tsuas yog ua los ntawm ib tug kws kho mob tshwj xeeb raws li txoj kev kuaj mob, kev ntsuam xyuas, kev tshawb fawb txog kev ua haujlwm.
Rau qhov kev npaj ntawm txoj kev kho mob, cov tshuaj nram qab no yog siv:
- Heptral ;
- Ursosan;
- Rifaren;
- Ursofalk;
- Naloxone;
- Rifampicin;
- ursodeoxycholic acid;
- Metronidazole;
- Rifadine;
- Nalmefer;
- Flumecinol;
- Cholestyramine;
- Bilignin;
- Zixorin;
- Ondansetron.
Nws kuj yog ib qho tseem ceeb los soj ntsuam kev noj haus nrog kev txwv ntawm cov tsiaj rog, noj cov vitamins.